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Polymorphic Ventricular Tachycardia

Polymorphic ventricular tachycardia

Ventricular Tachycardia

Ectopic ventricular focus, ventricular tachycardia

Basic Classification


Monomorphic Ventricular Tachycardia

Myocardial scarring, ectopic focus - reentry, Monomorphic ventricular tachycardia
  • Monomorphic VT is the most common type of ventricular tachycardia
  • Occurs in a structurally damaged heart
  • A reentry circuit forms in the scar
    • And generates impulses with a frequency of > 100/min.

  • All QRS complexes are monomorphic (identical)
    • Therefore, it is referred to as monomorphic


ECG single focus, single exit site, monomorphic ventricular tachycardia

Monomorphic Ventricular Tachycardia


Polymorphic Ventricular Tachycardia

  • Most commonly occurs during myocardial infarction
  • Polymorphic VT more frequently progresses to ventricular fibrillation (compared to monomorphic VT)
  • The mechanism is most often re-entry
  • Torsades de Pointes
  • QRS complexes are polymorphic (changing width and amplitude)
    • In the ventricles, there are usually at least 3 ectopic foci
      • Each activates the ventricles in a different direction (hence the change in shape of the QRS complexes)
  • According to the number of foci in the ventricles, we recognize 3 mechanisms of polymorphic ventricular tachycardia
    • Multifocal
    • Bifocal
    • Unifocal

Multifocal Polymorphic VT

ECG polymorphic ventricular tachycardia, multiple focus, multiple exit sites

Bifocal Polymorphic VT

ECG polymorphic ventricular tachycardia bifocal, Bidirectional ventricular tachycardia

Unifocal Polymorphic VT

ECG polymorphic ventricular tachycardia, single focus, multiple port of exits sites

Classification of Polymorphic Ventricular Tachycardia


ECG and Polymorphic Ventricular Tachycardia



Polymorphic ventricular tachycardia, single focus, multiple exit sites
ECG polymorphic ventricular tachycardia

Polymorphic Ventricular Tachycardia

  • In the ventricles, there are at least 3 ectopic foci (most commonly)
  • Width and amplitude of QRS complexes change
    • Features of ventricular tachycardia may be present
  • The mechanism is most commonly triggered activity (Torsades de Pointes)
  • Frequency is 100-300/min. (280/min. in this ECG)
  • The heart still functions as a pump
    • At frequencies > 160/min., it gradually stops functioning as a pump

Polymorphic VT and Ventricular Fibrillation


ventricular fibrillation
ECG ventricular fibrillation, Chaotic irregular deflections of varying amplitude

Ventricular Fibrillation



ECG polymorphic VT, Torsades de Pointes, Phenomenon R on T, Prolonged QT

Polymorphic Ventricular Tachycardia (Torsades de Pointes)



ECG Polymorphic VT, Torsades de Pointes (twisting morphology), Long QT interval, hypokalemia, phenomenon R on T

Polymorphic Ventricular Tachycardia (Torsades de Pointes)



ECG polymorphic VT after inferior STEMI infarction, AV dissociation

Polymorphic Ventricular Tachycardia



ECG Long QT interval, Phenomenon R on T, Torsades de Pointes (twisting morphology), Ventricular fibrillation

Polymorphic Ventricular Tachycardia (Torsades de Pointes) and Ventricular Fibrillation



ECG Bidirectional Ventricular Tachycardia, axis shifts 180 degrees, severe digoxin toxicity

Polymorphic Ventricular Tachycardia (Bidirectional Ventricular Tachycardia)




Sources

  • ECG from Basics to Essentials Step by Step
  • litfl.com
  • ecgwaves.com
  • metealpaslan.com
  • medmastery.com
  • uptodate.com
  • ecgpedia.org
  • wikipedia.org
  • Strong Medicine
  • Understanding Pacemakers





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Polymorphic Ventricular Tachycardia

Polymorphic ventricular tachycardia

Ventricular Tachycardia



Ectopic ventricular focus, ventricular tachycardia

Basic Classification


Monomorphic Ventricular Tachycardia

  • Monomorphic VT is the most common type of ventricular tachycardia
  • Occurs in a structurally damaged heart
  • A reentry circuit forms in the scar
    • And generates impulses with a frequency of > 100/min.

  • All QRS complexes are monomorphic (identical)
    • Therefore, it is referred to as monomorphic

Myocardial scarring, ectopic focus - reentry, Monomorphic ventricular tachycardia


ECG single focus, single exit site, monomorphic ventricular tachycardia

Monomorphic Ventricular Tachycardia

  • Frequency: 170/min.
  • Identical and Wide QRS Complexes (> 0.12s)
    • A re-entry impulse always exits in the same direction
      • Therefore, all QRS complexes are identical
  • P Waves are not visible
    • They are hidden within the wide QRS complexes
    • And the atria are activated retrogradely

Polymorphic Ventricular Tachycardia

  • Most commonly occurs during myocardial infarction
  • Polymorphic VT more frequently progresses to ventricular fibrillation (compared to monomorphic VT)
  • The mechanism is most often re-entry
  • Torsades de Pointes
  • QRS complexes are polymorphic (changing width and amplitude)
    • In the ventricles, there are usually at least 3 ectopic foci
      • Each activates the ventricles in a different direction (hence the change in shape of the QRS complexes)
  • According to the number of foci in the ventricles, we recognize 3 mechanisms of polymorphic ventricular tachycardia
    • Multifocal
    • Bifocal
    • Unifocal


ECG polymorphic ventricular tachycardia, multiple focus, multiple exit sites

Multifocal Polymorphic VT



ECG polymorphic ventricular tachycardia bifocal, Bidirectional ventricular tachycardia

Bifocal Polymorphic VT



ECG polymorphic ventricular tachycardia, single focus, multiple port of exits sites

Unifocal Polymorphic VT


Classification of Polymorphic Ventricular Tachycardia


ECG and Polymorphic Ventricular Tachycardia



Polymorphic ventricular tachycardia, single focus, multiple exit sites ECG polymorphic ventricular tachycardia

Polymorphic Ventricular Tachycardia

  • In the ventricles, there are at least 3 ectopic foci (most commonly)
  • Width and amplitude of QRS complexes change
    • Features of ventricular tachycardia may be present
  • The mechanism is most commonly triggered activity (Torsades de Pointes)
  • Frequency is 100-300/min. (280/min. in this ECG)
  • The heart still functions as a pump
    • At frequencies > 160/min., it gradually stops functioning as a pump

Polymorphic VT and Ventricular Fibrillation


ventricular fibrillation ECG ventricular fibrillation, Chaotic irregular deflections of varying amplitude

Ventricular Fibrillation

  • In the ventricles, there are multiple foci
    • The mechanism is most commonly micro re-entry
  • Frequency is 300-450/min.
  • Width and amplitude of QRS complexes change
    • QRS complexes are low, and their amplitude is not precisely defined
  • The heart does not function as a pump
    • At such a high frequency, diastole is ineffective
    • This is the basic difference from polymorphic VT
  • Signs of ventricular tachycardia are not present
  • QRS complexes decrease in amplitude and approximately 2 minutes later asystole occurs


ECG polymorphic VT, Torsades de Pointes, Phenomenon R on T, Prolonged QT

Polymorphic Ventricular Tachycardia (Torsades de Pointes)



ECG Polymorphic VT, Torsades de Pointes (twisting morphology), Long QT interval, hypokalemia, phenomenon R on T

Polymorphic Ventricular Tachycardia (Torsades de Pointes)



ECG polymorphic VT after inferior STEMI infarction, AV dissociation

Polymorphic Ventricular Tachycardia



ECG Long QT interval, Phenomenon R on T, Torsades de Pointes (twisting morphology), Ventricular fibrillation

Polymorphic Ventricular Tachycardia (Torsades de Pointes) and Ventricular Fibrillation



ECG Bidirectional Ventricular Tachycardia, axis shifts 180 degrees, severe digoxin toxicity

Polymorphic Ventricular Tachycardia (Bidirectional Ventricular Tachycardia)




Sources

  • ECG from Basics to Essentials Step by Step
  • litfl.com
  • ecgwaves.com
  • metealpaslan.com
  • medmastery.com
  • uptodate.com
  • ecgpedia.org
  • wikipedia.org
  • Strong Medicine
  • Understanding Pacemakers